Two cannabinoid compounds could be used to block the virus entering human cells, according to new research from Oregon State University.
Scientists have found that compounds in cannabis may prevent the spread of Covid.
A team at Oregon State University has published new research that suggests two cannabinoid acid could prevent the virus from infecting human cells.
Experts believe it could open the door to new treatments and prevent the disease.
However, scientists have shown that smoking marijuana is not a good way to protect yourself.
Journal of Natural Products published a study that found that spike protein may be bound to cannabigerolic (CBGA), and thus limit its damage potential.
Experts say the compounds can be taken orally, and they are also found in hemp.
Scientists have said that they have found the alpha and beta versions of the virus can be stopped from infecting individuals.
However, further research is required to confirm that it works against other strains.
Richard van Breemen was the leader of the study. Statement : “These cannabinoid acids are abundant in hemp and in many hemp extracts.
“They aren’t controlled substances like THC, the psychoactive element in marijuana.
“And our research showed the hemp compounds were equally effective against variants of SARS-CoV-2, including variant B.1.1.7, which was first detected in the United Kingdom, and variant B.1.351, first detected in South Africa.”
According to the scientist, the treatment could also be used to reduce the duration of illness in those already infected.
He continued his statement: “Any part of the infection and replication cycle is a potential target for antiviral intervention, and the connection of the spike protein’s receptor binding domain to the human cell surface receptor ACE2 is a critical step in that cycle.
“That means cell entry inhibitors, like the acids from hemp, could be used to prevent SARS-CoV-2 infection and also to shorten infections by preventing virus particles from infecting human cells. They bind to the spike proteins so those proteins can’t bind to the ACE2 enzyme, which is abundant on the outer membrane of endothelial cells in the lungs and other organs.”